Search results for "anoma [mediation]"

showing 10 items of 20 documents

Peroxisome proliferator-activated receptor alpha deficiency impairs regulatory T cell functions: Possible application in the inhibition of melanoma t…

2016

International audience; Regulatory T (Treg) cells are important to induce and maintain immunological self-tolerance. Although the progress accomplished in understanding the functional mechanism of Treg cells, intracellular molecules that control the mechanisms of their suppressive capacity are still on investigation. The present study showed that peroxisome proliferator-activated receptor-alpha deficiency impaired the suppressive activity of Treg cells on CD4(+)CD25(-) and CD8(+) T cell proliferation. In Treg cells, PPARα gene deletion also induced a decrease of migratory abilities, and downregulated the expression of chemokine receptors (CCR-4, CCR-8 and CXCR-4) and p27(KIP1) mRNA. Treg ce…

0301 basic medicineMaleAdoptive cell transferMESH: Tumor BurdenB16 melanoma tumorMelanoma ExperimentalMESH: T-Lymphocyte SubsetsCD4(+)CD25(+) regulatory T cellsBiochemistryMESH: Mice KnockoutImmunotherapy AdoptiveT-Lymphocytes RegulatoryPPARαMESH : T-Lymphocytes RegulatoryCell MovementT-Lymphocyte SubsetsMESH: Reverse Transcriptase Polymerase Chain ReactionMESH : Cell ProliferationMESH : Cell MovementMESH: AnimalsIL-2 receptorMESH: PPAR alphaMESH: Cell MovementCells CulturedMice KnockoutMESH : Melanoma ExperimentalbiologyMESH : Tumor BurdenReverse Transcriptase Polymerase Chain ReactionMESH : Reverse Transcriptase Polymerase Chain ReactionFOXP3hemic and immune systemsGeneral MedicineMESH: Gene Expression Regulation Neoplastic3. Good healthTumor BurdenMESH: Melanoma ExperimentalDNA-Binding ProteinsGene Expression Regulation Neoplasticmedicine.anatomical_structureMESH: Immunotherapy AdoptiveReceptors ChemokineMESH : DNA-Binding ProteinsMESH: Cells Culturedmedicine.medical_specialtyMESH : Receptors ChemokineMESH: Cell Line TumorRegulatory T cellMESH : Gene Expression Regulation NeoplasticT cellMESH : MaleMESH : PPAR alphachemical and pharmacologic phenomenaMESH : Mice Inbred C57BLMESH : Clonal Anergy03 medical and health sciencesMESH: Mice Inbred C57BLInternal medicineMESH: Cell ProliferationCell Line TumorMESH : Cells CulturedmedicineAnimals[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyPPAR alpha[ SDV.BBM ] Life Sciences [q-bio]/Biochemistry Molecular BiologyCell ProliferationClonal AnergyPerforinMESH : Cell Line TumorMESH: T-Lymphocytes RegulatoryMolecular biologyMESH: MaleMESH : T-Lymphocyte SubsetsGranzyme BMice Inbred C57BL030104 developmental biologyEndocrinologyPerforinMESH: Clonal Anergybiology.proteinMESH : Mice KnockoutMESH : AnimalsMESH: Receptors ChemokineCD8MESH: DNA-Binding ProteinsMESH : Immunotherapy Adoptive
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Melanoma-Nevus Discrimination Based on Image Statistics in Few Spectral Channels

2016

The purpose of this paper is to offer a method for discrimination of cutaneous melanoma from benign nevus, founded on analysis of skin lesion image. At the core of method is calculation of mean and standard deviation of pixel optical density values for a few narrow spectral bands. Calculated values are compared with discriminating thresholds derived from a set of images of benign nevi and melanomas with known diagnosis. Classification is done applying weighted majority rule to results of thresholding. Verification against the available multispectral images of 32 melanomas and 94 benign nevi has shown that the method using three spectral bands provided zero false negative and four false posi…

0301 basic medicineNevi and melanomasContextual image classificationImage classificationmelanoma detection.Multispectral imageSpectral bandsbiomedical optical imagingmedicine.disease01 natural sciencesThresholdingStandard deviation010104 statistics & probability03 medical and health sciences030104 developmental biologyCutaneous melanomaStatisticsmultispectral imagingmedicineNevus0101 mathematicsElectrical and Electronic EngineeringMathematicsElektronika ir Elektrotechnika
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Melanomas resist T-cell therapy through inflammation-induced reversible dedifferentiation.

2012

Adoptive cell transfer therapies (ACTs) with cytotoxic T cells that target melanocytic antigens can achieve remissions in patients with metastatic melanomas, but tumours frequently relapse. Hypotheses explaining the acquired resistance to ACTs include the selection of antigen-deficient tumour cell variants and the induction of T-cell tolerance. However, the lack of appropriate experimental melanoma models has so far impeded clear insights into the underlying mechanisms. Here we establish an effective ACT protocol in a genetically engineered mouse melanoma model that recapitulates tumour regression, remission and relapse as seen in patients. We report the unexpected observation that melanoma…

Adoptive cell transfermedicine.medical_treatmentCellular differentiationT cellBiologyProinflammatory cytokineMiceAntigenCell Line TumormedicineTumor MicroenvironmentCytotoxic T cellAnimalsHumansMelanomaCell ProliferationInflammationMultidisciplinaryTumor Necrosis Factor-alphaMelanomaCell DifferentiationImmunotherapyCell Dedifferentiationmedicine.diseaseAdoptive TransferMice Inbred C57BLDisease Models Animalmedicine.anatomical_structureImmunologyImmunotherapyNeoplasm TransplantationT-Lymphocytes Cytotoxicgp100 Melanoma AntigenNature
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Rapid High Efficiency Sensitization of CD8+ T Cells to Tumor Antigens by Dendritic Cells Leads to Enhanced Functional Avidity and Direct Tumor Recogn…

2003

Abstract Myeloid-origin dendritic cells (DCs) can develop into IL-12-secreting DC1 or non-IL-12-secreting DC2 depending on signals received during maturation. Through rapid culture techniques that prepared either mature, CD83+ DC1 or DC2 from CD14+ monocytes in only 2 days followed by a single 6–7 day DC-T cell coculture, we sensitized normal donor CD8+ T cells to tumor Ags (HER-2/neu, MART-1, and gp100) such that peptide Ag-specific lymphocytes constituted up to 16% of the total CD8+ population. Both DC1 and DC2 could sensitize CD8+ T cells that recognized peptide-pulsed target cells. However, with DC2, a general decoupling was observed between recognition of peptide-pulsed T2 target cells…

AdultCytotoxicity ImmunologicMaleReceptor ErbB-2CD8 AntigensT cellImmunologyAntigen presentationEpitopes T-LymphocyteStreptamerCD8-Positive T-LymphocytesBiologyLymphocyte ActivationInterleukin 21MART-1 AntigenAdjuvants ImmunologicAntigens NeoplasmT-Lymphocyte SubsetsCell Line TumorCell AdhesionmedicineHumansImmunology and AllergyCytotoxic T cellIL-2 receptorAntigen-presenting cellMelanomaCells CulturedAntigen PresentationMembrane GlycoproteinsCell DifferentiationDendritic CellsInterleukin-12Peptide FragmentsNeoplasm ProteinsCell biologymedicine.anatomical_structureCulture Media ConditionedImmunologyInterleukin 12FemaleImmunizationgp100 Melanoma AntigenThe Journal of Immunology
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Primary oral melanoma : a histopathological and immunohistochemical study of 22 cases of Latin America

2011

Objective: The aim of this study was to analyze the histopathological and immunohistochemical characteristics of 22 cases of primary oral melanomas (OM). Study Design: Twenty two cases of primary oral melanoma were analyzed by description of their histopathological features and immunohistochemical study using the antibodies S-100, HMB-45, Melan-A and Ki-67. Results: The mean age was 58 years and 14 cases were female. The main affected sites were the hard palate, followed by the upper gingiva. Microscopically, 15 cases presented level III of invasion, 2 cases were amelanotic and 13 showed a mixed epithelioid and plasmacytoid or spindle cells composition. Some cases showed necrosis, perivascu…

AdultMalePathologymedicine.medical_specialtyNecrosisProliferative indexPerineural invasionYoung AdultMART-1 AntigenmedicineHumansGeneral DentistryMelanomaAgedMouth neoplasmAged 80 and overOral Medicine and Pathologybusiness.industryMelanomaS100 ProteinsMiddle Agedmedicine.disease:CIENCIAS MÉDICAS [UNESCO]Immunohistochemistrymedicine.anatomical_structureKi-67 AntigenLatin AmericaOtorhinolaryngologyUNESCO::CIENCIAS MÉDICASImmunohistochemistrySurgeryHistopathologyResearch-ArticleFemaleMouth NeoplasmsHard palatemedicine.symptombusinessMelanoma-Specific Antigensgp100 Melanoma Antigen
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Synthesis, chemical characterization and preliminary in vitro antitumor activity evaluation of new ruthenium(II) complexes with sugar derivatives

2011

Abstract Three new complexes of Ru(II), namely [RuCl 2 (Glun-N,O) 2 ]Na 2 ( I ; Glun = glucosaminate), [RuCl 2 (1-Tglu)(EtOH) 2 ]Na ( II ; 1-Tglu = 1-thio-β- d -glucose) and [Ru 2 (EtOH) 6 (AL)Cl 4 ] ( III ; AL = 6′-aminolactose) were prepared from the same Ru(II) precursor, [RuCl 2 (DMSO) 4 ] (DMSO = dimethyl sulfoxide). The characterization of the complexes was carried out by elemental analysis, FT-IR, ES-MS, NMR, EXAFS and DFT calculations. The effectiveness of the complexes on metastatic melanoma A 375 was investigated. The results show that complex II is the most active species.

Antitumor activityCarbohydrates Anti-cancer Melanoma A375Extended X-ray absorption fine structureMetastatic melanomaStereochemistrychemistry.chemical_elementSulfoxideMedicinal chemistryIn vitroRutheniumInorganic ChemistrySugar derivativeschemistry.chemical_compoundchemistryElemental analysisMaterials ChemistryPhysical and Theoretical Chemistry
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Cytolytic T cell reactivity against melanoma-associated differentiation antigens in peripheral blood of melanoma patients and healthy individuals

1996

Antigenic peptides derived from several differentiation antigens of the melanocyte lineage were recently identified in human melanomas as targets for HLA-A2.1-restricted cytotoxic T lymphocytes (CTLs). To examine their potential role in tumour-directed immune responses in vivo, we determined CTL reactivity against seven antigenic peptides derived from the Melan A/MART-1, tyrosinase and gp100/Pmel17 antigens in the peripheral blood of 10 HLA-A2+ healthy controls and 26 HLA-A2+ melanoma patients. The influenza matrix peptide (GILGFVFTL) presented by HLA-A2.1 was used as a control peptide. CTL reactivity was assessed in a mixed lymphocyte 'peptide' culture assay. Reactivity against Melan A/MAR…

Cancer ResearchLymphocyte10050 Institute of Pharmacology and Toxicology610 Medicine & healthDermatologyPeripheral blood mononuclear cell2708 DermatologyMART-1 AntigenImmune systemMelanocyte differentiationAntigenAntigens NeoplasmTumor Cells CulturedmedicineHumansCytotoxic T cell1306 Cancer ResearchAmino Acid SequenceMelanomaneoplasmsMembrane GlycoproteinsMonophenol Monooxygenasebusiness.industryMelanomaProteinsmedicine.diseaseAntigens DifferentiationNeoplasm ProteinsCTL*medicine.anatomical_structureOncologyImmunology570 Life sciences; biology2730 OncologybusinessT-Lymphocytes Cytotoxicgp100 Melanoma AntigenMelanoma Research
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Temozolomide- and fotemustine-induced apoptosis in human malignant melanoma cells: response related to MGMT, MMR, DSBs, and p53

2009

Malignant melanomas are highly resistant to chemotherapy. First-line chemotherapeutics used in melanoma therapy are the methylating agents dacarbazine (DTIC) and temozolomide (TMZ) and the chloroethylating agents BCNU and fotemustine. Here, we determined the mode of cell death in 11 melanoma cell lines upon exposure to TMZ and fotemustine. We show for the first time that TMZ induces apoptosis in melanoma cells, using therapeutic doses. For both TMZ and fotemustine apoptosis is the dominant mode of cell death. The contribution of necrosis to total cell death varied between 10 and 40%. The O(6)-methylguanine-DNA methyltransferase (MGMT) activity in the cell lines was between 0 and 1100 fmol m…

Cancer ResearchProgrammed cell deathDNA repairDacarbazineBlotting WesternApoptosistemozolomideBiologyCollagen Type XIDNA Mismatch RepairNecrosisGliomaAntineoplastic Combined Chemotherapy ProtocolsTumor Cells CulturedmedicineHumansDNA Breaks Double-StrandedEverolimusPhosphorylationDNA Modification MethylasesMelanomaneoplasmsSirolimusTemozolomideTumor Suppressor ProteinsMelanomafotemustinemelanoma therapymedicine.diseaseDacarbazineEnzyme Activationmismatch repairDNA Repair EnzymesOncologyApoptosisCaspasesCancer researchFotemustineTumor Suppressor Protein p53Translational TherapeuticsMGMTmedicine.drugBritish Journal of Cancer
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Generation of CD8+T cells expressing two additional T-cell receptors (TETARs) for personalised melanoma therapy

2015

Adoptive T-cell therapy of cancer often fails due to the tumor cells' immune escape mechanisms, like antigen loss or down-regulation. To anticipate immune escape by loss of a single antigen, it would be advantageous to equip T cells with multiple specificities. To study the possible interference of 2 T-cell receptors (TCRs) in one cell, and to examine how to counteract competing effects, we generated TETARs, CD8(+) T cells expressing two additional T-cell receptors by simultaneous transient transfection with 2 TCRs using RNA electroporation. The TETARs were equipped with one TCR specific for the common melanoma antigen gp100 and one TCR recognizing a patient-specific, individual mutation of…

Cancer ResearchSkin Neoplasmsmedicine.medical_treatmentReceptors Antigen T-CellCD8-Positive T-LymphocytesBiologyImmunotherapy AdoptiveAntigenCell Line TumormedicineHumansCytotoxic T cellPrecision MedicineAntigen-presenting cellReceptorMelanomaPharmacologyT-cell receptorImmunotherapyResearch PapersMolecular biologyOncologyCancer researchMolecular MedicineCytokine secretionChaperonin Containing TCP-1CD8gp100 Melanoma AntigenCancer Biology & Therapy
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Clinical‑dermoscopic similarities between atypical nevi and early stage melanoma

2021

Atypical (Clark) nevi are benign tumors that may be considered precursors of melanoma. Many studies acknowledge a linear progression from typical to atypical nevi that eventually transform into melanoma. It is often challenging to differentiate a Clark nevus from melanoma, especially in its early stages, due to their clinical, dermoscopic, and histological resemblance. Dermoscopy is a powerful tool in early melanoma diagnosis, but it is a subjective method of examination. Therefore, the use of dermoscopic algorithms and checklists can overcome this issue. In the case of a difficult diagnosis, since both dermoscopy and histopathological exam are subjective methods of examination, modern mole…

Cancer Researchmedicine.medical_specialtyClark Nevusbusiness.industryMelanomaCancerArticlesGeneral Medicinemedicine.diseaseAtypical nevusDermatologyhistologyatypical neviImmunology and Microbiology (miscellaneous)melanomamedicinedermoscopyskin and connective tissue diseasesbusinessneoplasmsMelanoma diagnosisStage melanomaearly diagnosisExperimental and Therapeutic Medicine
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